Rewards include resistance to dewetting, compatibility with 100% aqueous cellular phases, different selectivity to HALO C18 and lengthier retention of polar compounds than classic C18 phases less than most cellular stage situations.

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Therapeutic vaccine candidates using viral vectors encoding LMP2 have efficiently induced virus-distinct CD8⁺ T cell responses in early-stage medical trials. In addition, adoptive T mobile therapies like posoleucel also focus on LMP2 alongside other latency proteins for instance EBNA1, reinforcing its importance being a conserved and immunodominant antigen.

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Among the these latency proteins, EBNA-3a plays a supportive but non-critical function in B mobile transformation: EBV mutants lacking EBNA-3a can still change B cells, but with considerably lessened effectiveness.

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